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                <text>Hacia un modelo experimental de ROP. Estudio estructural y ultraestructural /  M. Rey-Funes [et al.].</text>
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                <text>vol. 79, n. 1 (marzo-abril 2008), p. 20-32. Título: Archivos de Oftalmología de Buenos Aires</text>
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                <text>Retinopatia de la prematuridad</text>
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                <text>Hacia una integración orgánica /  R. Dodds.</text>
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                <text>vol. 14, n. 2,  (mayo 2001), p. 8-9Título: MO Médico Oftalmólogo</text>
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                <text>Hallazgos clínicos y genéticos en una familia con retinosis pigmentaria recesiva ligada al X.</text>
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                <text>Presentamos el caso clínico de una familia con diagnóstico de retinosis pigmentaria con herencia recesiva ligada al X. Las mutaciones que afectan al gen regulador de la GTPasa de la retinosis pigmentaria (RPGR) son la causa más común de RP recesiva ligada al cromosoma X. El fenotipo es uno de los más graves entre todas las formas de RP, característico por su inicio temprano y rápida progresión a la ceguera en personas jóvenes.</text>
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                <text>Archivos Argentinos de Oftalmología, nro. 19, diciembre de 2021, p. 43-52.</text>
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                <text>vol. 77, nº 2 (oct-dic 2004), p. 212-221 Título: Archivos de Oftalmología de Buenos Aires|773x|0066-6777</text>
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                <text>Objetivo. La glomerulopatía proliferativa mesangial (GPM), se caracteriza por la proliferación de las células mesangiales con acumulación de matriz mesangial extracelular.&#13;
&#13;
El objetivo del presente artículo es describir  los hallazgos retinales en el fondo de ojo de una paciente con GPM.&#13;
&#13;
Caso clínico. Una paciente de 34 años concurrió para realizar control de rutina. Tenía antecedentes de asma bronquial, hipertensión arterial (HTA) y GPM con depósitos de IgM que requirió trasplante renal. En su exploración se constató una agudeza visual de 10/10 en ambos ojos. Como hallazgo, en el fondo de ojo (FO) se detectaron múltiples lesiones redondeadas de menos de ¼ de diámetro papilar en polo posterior con compromiso macular, perifoveales y en retina periférica 360°. En la tomografía de coherencia óptica, se observaron alteraciones del perfil posterior compatibles con depósitos drusenoides a nivel del epitelio pigmentario retinal (EPR) y como múltiples imágenes hiperreflectivas por alteración del EPR a nivel de la retinofluoresceinografía. &#13;
&#13;
Se realizaron además estudios de campo visual que evidenciaron escotomas aislados en ambos ojos y electroretinograma de características normales.&#13;
&#13;
Conclusión. Como hemos visto en el presente caso, los hallazgos retinales en la GPM brindan información sobre la afectación ocular de la glomerulopatía proliferativa mesangial y nos permiten realizar el seguimiento de estos pacientes para la detección temprana y/o prevención de sus posibles complicaciones.</text>
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                <text>Oftalmología Clínica y Experimental, vol. 18, nro. 3, sep. de 2025, p. 244-350</text>
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                <text>Halos y visión borrosa /  A. Gómez y otros.</text>
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                <text>vol. 24, n. 2 (jun. 2011), p. 44-5. Título: Médico Oftalmólogo</text>
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